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Genevention will help to systematically understand APC stop codon mutation readthrough

In these mutations, a codon that codes for an amino acid is changed into a stop codon, preventing translation of the complete, functional protein. This type of mutation can be found in nearly all known monogenetic diseases including most rare diseases, summing up to 5,000 – 10,000 diseases or tens of million patients worldwide. It is a viable yet challenging therapeutic strategy to treat these PTCs with translational read­through-inducing drugs (TRIDs).

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1. Realistic in silico generation and augmentation of single cell RNA-seq data using Generative Adversarial Neural Networks

2. Retinoic acid-induced expression of Hnf1b and Fzd4 is required for pancreas development in Xenopus laevis.

3. Critical Assessment of Metagenome Interpretation-a benchmark of metagenomics software.

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